Temat: liver parenchyma - Katalog OPAC zbiorów

Skocz do pozycji: 1.

Tytuł:: Comparative ultrastructural studies of the alterations to mouse hepatic parenchyma during Trichinella spiralis or Toxocara canis infection
Autorzy:: Dabrowska, J.
Dybicz, M.
Powiązania:: https://bibliotekanauki.pl/articles/6278.pdf
Data publikacji:: 2016
Wydawca:: Polskie Towarzystwo Parazytologiczne
Tematy:: ultrastructure
alteration
mouse
mice
hepatic parenchyma
parenchyma
Trichinella spiralis
Toxocara canis
infection
pathological change
liver
Źródło:: Annals of Parasitology; 2016, 62, Suppl.
0043-5163
Pojawia się w:: Annals of Parasitology
Dostawca treści:: Biblioteka Nauki

Artykuł

Skocz do pozycji: 2.

Tytuł:: Histopathologic and immunohistochemical lesions in liver of mink infected with Aleutian disease virus
Autorzy:: Valdovska, A.
Pilmane, M.
Powiązania:: https://bibliotekanauki.pl/articles/30104.pdf
Data publikacji:: 2011
Wydawca:: Polska Akademia Nauk. Czytelnia Czasopism PAN
Tematy:: Aleutian disease
Aleutian mink disease parvovirus
animal infection
beta-defensin-2
histopathology
immunohistochemistry
liver
liver parenchyma
mink
nerve growth factor receptor
metalloproteinase
Opis:: Parvovirus of Aleutian disease causes mainly damage to kidneys, but immune complexes deposition and damage may occur also in other organs. In mink farms of Latvia the liver dystrophy or hepatic lipidosis of mink is widely distributed. The goal of this study was to examine probability of liver damage and regeneration of mink infected with Aleutian disease virus. Liver injury was assessed histologically. The mink liver demonstrated inflammation of liver parenchyma and foci of fatty liver. In immunohistochemistry, during liver regeneration the matrix metalloproteinases MMP-9, vascular endothelial growth factor and β-defensin 2 expressions were lower, but MMP-2 and nerve growth factor receptor p75 expression was increased.
Źródło:: Polish Journal of Veterinary Sciences; 2011, 14, 1
1505-1773
Pojawia się w:: Polish Journal of Veterinary Sciences
Dostawca treści:: Biblioteka Nauki

Artykuł

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