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    Wyświetlanie 1-3 z 3
    Tytuł:
    Giant cell tumor of the frontal bone – case report
    Guz olbrzymiokomórkowy kości czołowej - opis przypadku
    Autorzy:
    Zagacki, Dawid
    Braun, Marcin
    Zbróg, Zbigniew
    Morawiec-Sztandera, Alina
    Kaczmarczyk, Dariusz
    Powiązania:
    https://bibliotekanauki.pl/articles/1399455.pdf
    Data publikacji:
    2019
    Wydawca:
    Index Copernicus International
    Tematy:
    chronic kidney disease
    frontal bone tumor
    guz kości czołowej
    przewlekła niewydolność nerek
    Opis:
    Immunosuppression is a characteristic feature of chronic kidney disease. It may lead to the increased risk of infections and neoplastic diseases occurrence. We present an unusual case of 57-years-old women with uncommon neoplasm of the head and neck region – osteoclastoma.
    Cechą charakterystyczną w przypadku przewlekłej niewydolności nerek jest obniżenie odporności pacjenta, co prowadzi do zwiększonego ryzyka wystąpienia zakażeń czy chorób nowotworowych. Prezentujemy przypadek 57-letniej kobiety z rozpoznanym rzadkim nowotworem regionu głowy i szyi – guzem olbrzymiokomórkowym.
    Źródło:
    Polski Przegląd Otorynolaryngologiczny; 2019, 8, 2; 54-56
    2084-5308
    2300-7338
    Pojawia się w:
    Polski Przegląd Otorynolaryngologiczny
    Dostawca treści:
    Biblioteka Nauki
    Artykuł
    Tytuł:
    Red blood cell and plasma glutathione peroxidase activities and selenium concentration in patients with chronic kidney disease: A review
    Autorzy:
    Zachara, Bronisław
    Gromadzińska, Jolanta
    Wąsowicz, Wojciech
    Zbróg, Zbigniew
    Powiązania:
    https://bibliotekanauki.pl/articles/1041155.pdf
    Data publikacji:
    2006
    Wydawca:
    Polskie Towarzystwo Biochemiczne
    Tematy:
    hemodialysis
    selenium
    kidney transplantation
    glutathione peroxidase
    chronic kidney disease
    antioxidants
    plasma
    Opis:
    The metabolism of oxygen in aerobic organisms leads to generation of reactive oxygen species (ROS). These entities are able to oxidize almost all classes of macromolecules, including proteins, lipids and nucleic acids. The physiological level of ROS is usually regulated by antioxidant defense mechanisms. There are at least three groups of antioxidant enzymes: superoxide dismutases, catalases and glutathione peroxidases (GSH-Pxs) which neutralize ROS. The trace elements (copper, zinc and selenium) bound to the active sites of the above listed enzymes play an important role in the antioxidant defense system. In mammals, a major function of selenium (Se) and Se-dependent GSH-Pxs is to protect cells from oxidative stress. Selenium concentrations and GSH-Px activities are altered in blood components of chronic kidney disease (CKD) patients. The Se level is frequently lower than in healthy subjects and the concentration very often decreases gradually with advancing stage of the disease. Studies on red cell GSH-Px activity in CKD patients reported its values significantly lower, significantly higher and lower or higher, but not significantly as compared with healthy subjects. On the other hand, all authors who studied plasma GSH-Px activity have shown significantly lower values than in healthy subjects. The degree of the reduction decreases gradually with the progression of the disease. High inverse correlations were seen between plasma GSH-Px activity and creatinine level. A gradual decrease in plasma GSH-Px activity in CKD patients is due to the fact that this enzyme is synthesized predominantly in the kidney and thus the impairment of this organ is the cause of the enzyme's lower activity. Se supplementation to CKD patients has a slightly positive effect in the incipient stage of the disease, but usually no effect was observed in end-stage CKD. Presently, kidney transplantation is the only treatment that may restore plasma Se level and GSH-Px activity in patients suffering from end-stage CKD. A few studies have shown that in kidney recipients, plasma Se concentration and GSH-Px activity are restored to normal values within a period of 2 weeks to 3 months following surgery and thus it can be acknowledged that Se supplementation to those patients has a positive effect on plasma GSH-Px activity.
    Źródło:
    Acta Biochimica Polonica; 2006, 53, 4; 663-677
    0001-527X
    Pojawia się w:
    Acta Biochimica Polonica
    Dostawca treści:
    Biblioteka Nauki
    Artykuł
    Tytuł:
    Selenium supplementation to chronic kidney disease patients on hemodialysis does not induce the synthesis of plasma glutathione peroxidase
    Autorzy:
    Zachara, Bronislaw
    Gromadzinska, Jolanta
    Zbrog, Zbigniew
    Swiech, Rafal
    Wasowicz, Wojciech
    Twardowska, Ewa
    Jablonska, Ewa
    Sobala, Wojciech
    Powiązania:
    https://bibliotekanauki.pl/articles/1040656.pdf
    Data publikacji:
    2009
    Wydawca:
    Polskie Towarzystwo Biochemiczne
    Tematy:
    hemodialysis
    selenium supplementation
    glutathione peroxidase
    chronic kidney disease
    plasma
    Opis:
    Background: Numerous authors have shown that selenium (Se) concentration and glutathione peroxidase (GSH-Px) activity in plasma of chronic kidney disease (CKD) patients are lower than in healthy subjects, but there are only few publications on the level of GSH-Px protein in those patients and no reports on the effect of Se supplementation to HD patients on the level of this enzyme. Subjects and Methods: Se concentration and GSH-Px protein level in plasma were measured in a group of 30 CKD patients on hemodialysis (HD) supplemented with 200 µg Se/day for 3 months, and 28 patients on HD administered with placebo. Se concentration was measured by graphite furnace atomic absorption spectrometry and plasma GSH-Px protein level by the sandwich ELISA method using polyclonal antibody specific for human plasma GSH-Px. Results: Se concentration in patients on placebo did not change throughout the 3-month study period, but increased significantly in Se supplemented group. Se supplementation to CKD patients on HD had no effect on the level of GSH-Px protein. Conclusions: The lack of GSH-Px protein in CKD patients on HD is not linked to Se deficiency since the level of this element increased after Se supplementation while enzyme protein level did not change. The damaged kidney of HD patients is unable to synthesize GSH-Px, even after induction with selenium.
    Źródło:
    Acta Biochimica Polonica; 2009, 56, 1; 183-187
    0001-527X
    Pojawia się w:
    Acta Biochimica Polonica
    Dostawca treści:
    Biblioteka Nauki
    Artykuł
      Wyświetlanie 1-3 z 3

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