Autor: Ostrowski, Jerzy - Katalog OPAC zbiorów

Tytuł:: Functions of tourist maps of towns as a basis for designing their content: an example of pragmatic approach to the map language
Autorzy:: Ostrowski, Jerzy
Ostrowski, Wiesław
Powiązania:: https://bibliotekanauki.pl/articles/1968258.pdf
Data publikacji:: 1988-06-01
Wydawca:: Uniwersytet Warszawski. Wydział Geografii i Studiów Regionalnych
Źródło:: Miscellanea Geographica. Regional Studies on Development; 1988, 3; 397-402
0867-6046
2084-6118
Pojawia się w:: Miscellanea Geographica. Regional Studies on Development
Dostawca treści:: Biblioteka Nauki

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Skocz do pozycji: 22.

Tytuł:: Wyznaczanie dynamicznej wytrzymałości piezoelektrycznych belek z wykorzystaniem akustycznych fal powierzchniowych
Autorzy:: Filipiak, Jerzy.
Ostrowski, Jerzy.
Kopycki, Cezary.
Powiązania:: Biuletyn Wojskowej Akademii Technicznej 1998, nr 7-8, s. 153-160
Data publikacji:: 1998
Tematy:: Fale akustyczne powierzchniowe
Radiokomunikacja wojskowa
Opis:: Rys.; bibliogr.; Abstr., Rez., streszcz.
Dostawca treści:: Bibliografia CBW

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Skocz do pozycji: 23.

Tytuł:: Increased expression of ribosomal protein S2 in liver tumors, posthepactomized livers, and proliferating hepatocytes in vitro.
Autorzy:: Kowalczyk, Piotr
Woszczyński, Marek
Ostrowski, Jerzy
Powiązania:: https://bibliotekanauki.pl/articles/1043722.pdf
Data publikacji:: 2002
Wydawca:: Polskie Towarzystwo Biochemiczne
Tematy:: ribosomal protein S2
partial hepatectomy
hepatocytes
hepatocellular carcinoma
Opis:: The ribosomal protein S2 (RPS2) is encoded by a gene from the highly conserved mammalian repetitive gene family LLRep3. It participates in aminoacyl-transfer RNA binding to ribosome, potentially affecting the fidelity of mRNA translation. These studies were designed to measure the expression of RPS2 during increased cell proliferation. Using Western and Northern blot analyses, we found that the levels of RPS2 protein and its corresponding mRNA were higher in mouse hepatocellular carcinoma, in mouse livers after one-third partial hepatectomy, and in serum-starved cultured hepatocytes following serum treatment. Our study shows that the increased expression of RPS2 correlates with increased cell proliferation. However, whether the altered expression of this protein reflects its involvement in cellular proliferation or represents an associated phenomena is still a key question that needs to be explored.
Źródło:: Acta Biochimica Polonica; 2002, 49, 3; 615-624
0001-527X
Pojawia się w:: Acta Biochimica Polonica
Dostawca treści:: Biblioteka Nauki

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Skocz do pozycji: 24.

Tytuł:: Expression of genes encoding mitochondrial proteins can distinguish nonalcoholic steatosis from steatohepatitis
Autorzy:: Bragoszewski, Piotr
Habior, Andrzej
Walewska-Zielecka, Bozena
Ostrowski, Jerzy
Powiązania:: https://bibliotekanauki.pl/articles/1041083.pdf
Data publikacji:: 2007
Wydawca:: Polskie Towarzystwo Biochemiczne
Tematy:: NAFLD
gene expression
NASH
mitochondria
Opis:: In patients without substantial alcohol use, triglyceride accumulation in the liver can lead to nonalcoholic fatty liver disease (NAFLD) that may progress to nonalcoholic steatohepatitis (NASH). The differential diagnosis between NAFLD and NASH can be accomplished only by morphological examination. Although the relationship between mitochondrial dysfunction and the progression of liver pathologic changes has been described, the exact mechanisms initiating primary liver steatosis and its progression to NASH are unknown. We selected 16 genes encoding mitochondrial proteins which expression was compared by quantitative RT-PCR in liver tissue samples taken from patients with NAFLD and NASH. We found that 6 of the 16 examined genes were differentially expressed in NAFLD versus NASH patients. The expression of hepatic HK1, UCP2, ME2, and ME3 appeared to be higher in NASH than in NAFLD patients, whereas HMGCS2 and hnRNPK expression was lower in NASH patients. Although the severity of liver morphological injury in the spectrum of NAFLD-NASH may be defined at the molecular level, expression of these selected 6 genes cannot be used as a molecular marker aiding histological examination. Moreover, it is still unclear whether these differences in hepatic gene expression profiles truly reflect the progression of morphological abnormalities or rather indicate various metabolic and hormonal states in patients with different degrees of fatty liver disease.
Źródło:: Acta Biochimica Polonica; 2007, 54, 2; 341-348
0001-527X
Pojawia się w:: Acta Biochimica Polonica
Dostawca treści:: Biblioteka Nauki

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Skocz do pozycji: 25.

Tytuł:: Barretts esophagus associates with a variant of IL23R gene
Autorzy:: Gaj, Pawel
Mikula, Michal
Wyrwicz, Lucjan
Regula, Jaroslaw
Ostrowski, Jerzy
Powiązania:: https://bibliotekanauki.pl/articles/1040756.pdf
Data publikacji:: 2008
Wydawca:: Polskie Towarzystwo Biochemiczne
Tematy:: Barrett's esophagus
risk
SNP
IL23R
association study
Opis:: Gastroesophageal reflux disease is regarded as a spectrum of diseases: non-erosive reflux disease (NERD), erosive reflux disease (ERD), and the far end of the spectrum represented by patients with Barrett's esophagus. Among predisposing factors, both risk and protective polymorphic variants of several genes may influence the clinical outcomes of reflux disease. Consequently, different molecular mechanisms are likely to underlie the development of clinical variants of reflux disease. Ninety six patients with reflux disease were screened for polymorphisms of CARD15, SLC22A4 (OCTN1), SLC22A5 (OCTN2), DLG5, ATG16L1 and IL23R genes which had previously been found to associate with immune-mediated chronic inflammatory disorders. While none of the polymorphisms were associated with NERD or ERD, the 1142G/A variant of the IL23R gene was found to be a risk variant in Barrett's esophagus patients. The IL23/IL23R pathway may modulate STAT3 transcriptional activity which is an essential regulator not only of immune-mediated inflammation, but also of inflammatory-associated apoptosis resistance. Although the mechanisms of metaplastic transition of inflamed squamous epithelium are undetermined as yet, our findings suggest potential involvement of alternations in the IL23/IL23R pathway as a molecular background of Barrett's esophagus development.
Źródło:: Acta Biochimica Polonica; 2008, 55, 2; 365-369
0001-527X
Pojawia się w:: Acta Biochimica Polonica
Dostawca treści:: Biblioteka Nauki

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