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Wyszukujesz frazę "Guria, Srikanta" wg kryterium: Autor


Wyświetlanie 1-3 z 3
Tytuł:
Lead (Pb) Induces Paraptosis Like Cell Death in Hemocytes of Lamellidens Sp: A Preliminary Study
Autorzy:
Guria, Srikanta
Powiązania:
https://bibliotekanauki.pl/articles/1156374.pdf
Data publikacji:
2018
Wydawca:
Przedsiębiorstwo Wydawnictw Naukowych Darwin / Scientific Publishing House DARWIN
Tematy:
Apoptosis
Cellular Death
Hemocytes
Lamellidens marginalis
Lead (Pb)
Molluscs
Paraptosis
Opis:
Molluscs represent a simple and significant model system to test how specific metals can simultaneously affect development and putative mechanisms of defense and/or cell death. Lamellidens marginalis (Bivalvia: Eulamellibranchiata) are the molluscs of Indian freshwater ecosystem and important biological resources. Lead (Pb) is widespread heavy metal that is released into the environment from different sources. Their accumulation in the soils can become dangerous to all kinds of organisms, including plants, animals and human life, causing many adverse effects. Lamellidens are used as reliable markers and bioindicators highly sensitive to environmental changes. The aim of the present work was to determine the toxic effect of lead on the cytomorphology of hemocytes of Lamellidens marginalis and to evaluate its potential as a bio monitor for detecting a heavy-metal polluted environment. Treated cells showed apoptosis and paraptosis (vacuolation) like features. Morphological analyses suggested an irretrievable destruction of normal morphology of hemocyte. However further studies are needed to investigate this topic, particularly in relation to the pollutants reported in this paper.
Źródło:
World Scientific News; 2018, 94, 2; 163-172
2392-2192
Pojawia się w:
World Scientific News
Dostawca treści:
Biblioteka Nauki
Artykuł
Tytuł:
Macrophages in Diabetes Mellitus: A Review on Understanding of Macrophage Function
Autorzy:
Chatterjee, Susraba
Guria, Srikanta
Powiązania:
https://bibliotekanauki.pl/articles/1059398.pdf
Data publikacji:
2019
Wydawca:
Przedsiębiorstwo Wydawnictw Naukowych Darwin / Scientific Publishing House DARWIN
Tematy:
Alloxan
Beta-cell Therapy
Cytokines
Diabetes mellitus
Diabetic Nephropathy
Diabetic Retinopathy
Macrophage
Macrophage Type 1
Macrophage Type 2
Streptozotocin
Opis:
Diabetes mellitus (DM) causes millions of deaths all over the world. Immune system contains macrophages that play very important role in DM. Excessive secretion of different cytokines can induce the DM development. Diabetes mellitus (DM) also affect the function of macrophage. We review the important findings regarding the role of macrophage in DM. This review may emphasize future direction towards development of novel immune-modulatory therapeutic intervention.
Źródło:
World Scientific News; 2019, 134, 2; 319-325
2392-2192
Pojawia się w:
World Scientific News
Dostawca treści:
Biblioteka Nauki
Artykuł
Tytuł:
Alteration of Cytomorphology of Peritoneal Macrophages of Albino Rat Exposed to Mercuric Compound
Autorzy:
Srikanta, Guria
Powiązania:
https://bibliotekanauki.pl/articles/1158129.pdf
Data publikacji:
2018
Wydawca:
Przedsiębiorstwo Wydawnictw Naukowych Darwin / Scientific Publishing House DARWIN
Tematy:
Apoptosis
Blebbing
Macrophage
Mercury
Rat
Opis:
Mercury is among the heavy metals that have been reported to cause devastating health problems worldwide. The present work was aimed at investigating the effects of mercury chloride on the cytomorphology of the peritoneal macrophages of rat. The current study characterizes the mechanism by which mercury, a toxic metal, induces death in rat macrophages. In mercury treated group significant numbers of peritoneal macrophages were found to be pyknotic. The cellular death was confirmed by membrane blebbing and membrane rupture. Percentages of cells showing membrane blebbing was increased significantly after treatment. The result indicated mercury induced toxicity may affect macrophage mediated immunity. So macrophage activation during heavy metal mediated reaction is an interesting area to be explored in future researches.
Źródło:
World Scientific News; 2018, 92, 2; 392-399
2392-2192
Pojawia się w:
World Scientific News
Dostawca treści:
Biblioteka Nauki
Artykuł
    Wyświetlanie 1-3 z 3

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