- Tytuł:
-
Wpływ roztworu kwasu solnego na inwazje Ascaris suum oraz aktywność enzymów trawiennych przewodu pokarmowego i surowicy prosiąt osesków
EFFECT OF HCI ADMINISTRATION ON THE INVASION OF ASCARIS SUUM AND THE ENZYME ACIIVITY INTO DIGESTIVE SYSTEM AND IN SERUM OF NEWBORN PIGLETS - Autorzy:
-
Jabłonowski, Z.
Romaniuk, K.
Piechocki, D.
Żółtowska, K.
Łukaszewicz-Babecka, J.
Dziekońska-Rynko, J. - Powiązania:
- https://bibliotekanauki.pl/articles/2149103.pdf
- Data publikacji:
- 1995
- Wydawca:
- Polskie Towarzystwo Parazytologiczne
- Tematy:
-
pasozyty zwierzat
przewod pokarmowy
enzymy trawienne
oseski
aktywnosc enzymatyczna
prosieta
kwas solny
zywiciele
surowica krwi
parazytologia
glista swinska
inwazja pasozytnicza
Ascaris suum - Opis:
- The studies were carried out on twenty newborn piglets. They were divided into four groups. The groups no 3 and no 4 were given intragastric 0.18% HCl from the 3th day of experiment. The groups no 2 and no 4 were infected on the 7th day with 10000 invasive eggs of Ascaris suum. The presence of A.suum larvae in the lungs and liver was examined after one week lasting invasion by BAERMANN rnethod. The total acidity in the gastric content was measured. The activity of alpha-amylase, lipase and proteases was determined in the extracts from pancreas and in the contents of stomach, duodenum and jejunum. The level of pepsinogens and alpha-amylase in the animals serum was studied. The intensity of Ascaris invasion was slightly higher in the group which was given HCl than in the infected group without HCl. The activity of digestive enzymes in the both groups was similar. Only in the stomach content from the 4th group the activity of pepsin was higher (p<0.05), and alpha-amylase and lipase were lower (p <0.01) than in the 2nd and in the 3th group. The level of pepsinogens was always lower and alpha-amylase higher in the serum of infected animals than in uninfected groups.
- Źródło:
-
Wiadomości Parazytologiczne; 1995, 41, 4; 413-419
0043-5163 - Pojawia się w:
- Wiadomości Parazytologiczne
- Dostawca treści:
- Biblioteka Nauki